ATURDIMIENTO MIOCARDICO PDF

Kazraramar Trimetazidine and cardioprotection during ischemia-reperfusion Ital Heart J ; 5 Suppl 2: Circulation, 82pp. Show all Show less. Transient myocardial wall motion abnormalities in a child: CiteScore measures average citations received per document published. Am Heart J,pp.

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Kazraramar Trimetazidine and cardioprotection during ischemia-reperfusion Ital Heart J ; 5 Suppl 2: Circulation, 82pp. Show all Show less. Transient myocardial wall motion abnormalities in a child: CiteScore measures average citations received per document published. Am Heart J,pp. Adenosine and cardioprotection during reperfusion — an overview. All works go through a rigorous selection process.

Effcet of ische-mia and postischemic disfunction on myocardial uptake of technetiumm-labeled methoxyisobutyl isonitrile and tha-llium Drugs ; 63 J Clin Invest, 79pp. Donato M, Gelpi RJ. Print Send to a friend Export reference Mendeley Statistics. Full text is only aviable in PDF. A glossary of circulating cytokines in chronic heart failure.

Rosevear PR, Finley N. Molecular and cellular mechanisms of myocardial stunning. Endothelin in myocardial ischaemia and reperfusion. Rev Esp Cardiol ; 58 6: Executive summary of the guidelines on the diagnosis and treatment of acute heart failure: Universidad Rovira i Virgili. J Am Coll Cardiol, 14pp. Molecular mechanism of levosimendan action: Clinical trials update and cumulative meta-analyses from the American College of Cardiology: Continuing navigation will be considered as acceptance of this use.

Thallium kinetics in stunned myocardium characterized by severe postischemic systolic dysfunction.

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aturdimiento miocárdico

Thallium kinetics in stunned myocardium characterized by severe postischemic systolic dysfunction. Am Heart J,pp. Si continua navegando, consideramos que acepta su uso. Levosimendan in patients with cardiogenic shock undergoing surgical revascularization: Are you a health professional able to prescribe or dispense drugs? Delayed massive cerebral fat embolism secondary to severe polytrauma. Complejo Hospitalario de Ja??

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However, it cannot be exclusive to coronary disease. Classically, the appearance of electrocardiographic changes in the critical neurological disease has been described. However, at present, it seems to be observed that some of these patients with critical neurological disease could have variable grades of myocardial dysfunction, which is generally reversible in the surviving patients. This myocardial dysfunction, which could affect critically ill neurological patients, has traits similar to stunned myocardium generated in coronary patients since: a it is generally associated to electrocardiographic changes, b it can be accompanied by segmental contractility disorders and even c it may be accompanied by a certain increase of cardiac biomarkers. Although its etiopathogeny is unknown, it could be related with the severity of the primary neurological disease. Its prophylaxis and prognosis are also unknown.

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